Genetics

...the odds against DNA assembling by chance are10⁴⁰:1
[according to Fred Hoyle, Evolution from Space,1981].

Introduction

The living cell provides our greatest example of intelligent design. Within its confines resides a molecular machinery largely beyond our comprehension. The complexity of the cell may very well exceed that of the universe outside of our world, and yet there are those who can believe it simply formed through random processes .

The scientific community has become largely atheistic and generally assumes that cells formed without God or intelligent design. Therefore existing genetic theories have been developed by those who do not believe the cell was designed, and are not looking for mechanisms that were made to intentionally produce the genetic changes which allow organisms to adapt. Secular science believes that evolution is largely the result of biochemical accidents. Although geneticists and breeders have thoroughly established that genetic recombination is responsible for the variations of plant and animal breeds, we are still taught that random mutations produced the natural varieties of species such as the finches on the Galapagos islands. This contrast between fact and teachings is the result of atheistic theoretic necessity, which must propose that random reactions unreliant upon living systems are responsible for evolution.

Mutations or Genetic Recombination?

There are two sources of genetic variability; genetic recombination and mutation. Mutations are random, unintentional nucleotide alterations that can occur in many ways, such as by errors during replication, or by exposure to chemical mutagens. Genetic recombination, on the otherhand, is performed intentionally by cellular machinery and its products remains largely uncharacterized. Both mutation and recombination can modify genes, but we are being incorrectly taught that mutation is the primary source of variability driving evolution.

Little is known about recombination, except that reactions occur between chromosomes, which alter the genome of each daughter cell so no two offspring are ever identical. Given our level of understanding, we can not yet place a limitation on their ability to manipulate DNA. It is clear the genetic constitution of organisms is not static, and the cell's molecular machinery is altering genes and creating new alleles with each passing generation. The purpose of these reactions is clear. They occur so that organisms would be able to adapt physically and biochemically, and thereby occupy earth's broad range of habitats.

Finches from the Galapagos It has been recognized for decades that the differences found among offspring from the same parents were the result of recombination events during meiosis. Domestic breeds, for example, are recombinants, not mutants. These rearrangements are being performed by design to provide evolutionary potential to all organisms. Contrary to popular beliefs, evolution through recombination is not random, but is instead highly systematic. In comparison, mutations are random destructive changes that destroy information. Mutations are most typically deleterious, disruptive to genome function, and corrected by the cell when detected. Evolution truly occurs through a history of genetic recombination and natural selection, but the way it is taught, you would never know anything but mutation was involved.

Conclusion

Contrary to what evolutionists teach, adaptation is the result of systematic and intentional cellular reactions. However, in contrast to typical creationist perceptions, this change is not limited to the variability originally possessed by the organism. God created a cellular machinery that is performing a level of self genetic engineering. This process is creating new information and able to modify organisms so dramatically we can frequently not recognize them as related. The physical manifestations of these reactions is hard to predict, and it is also quite possible that genetic editions are being made in direct response to environmental demands.

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Genetics

Introduction

Mutations or Genetic Recombination?

Conclusion

See Also